Educational only. Not medical advice. Invite-only research preview.No PHI. Do not share patient names or identifying information (HIPAA).
MytoIntelligence
All targets

Molecular target

5α-Reductase

Enzyme converting testosterone to dihydrotestosterone (DHT). Inhibition (finasteride, dutasteride) reduces DHT — the basis for benign prostatic hyperplasia and androgenetic alopecia therapy. Saw palmetto and several other plant extracts have weak 5α-reductase inhibition.

3 drugs act here4 plants reach it via their compounds

Educational use only. This page summarizes published research and traditional-use records for educational purposes. It does not diagnose, treat, cure, or prevent any disease. Do not start, stop, or change medications based on this information. Discuss any decisions about therapies — pharmaceutical or botanical — with a qualified clinician who knows your medical history.

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Pharmaceutical agents

Drugs that act on 5α-Reductase

These medications have 5α-Reductase among their molecular targets. Sharing a target is a mechanistic relationship — it does not make any plant below an alternative to, or substitute for, these drugs.

Botanical connections

Plants whose compounds act on 5α-Reductase

Each plant below contains a named compound documented to act on 5α-Reductase. The compound and the reason for the connection are shown on every edge — a shared mechanism, not a therapeutic equivalence.

  • Free fatty acids (lauric, myristic, oleic)Fatty acid mixture

    Lipophilic fraction of the berry; weak 5α-reductase inhibition. Preferential extraction by hexanic methods (Permixon) is the basis of standardized products.

  • β-SitosterolPhytosterol

    Plant sterol with 5α-reductase modulating activity; also found in pygeum, pumpkin seed, and many other plants.

  • β-SitosterolPhytosterol

    Same mechanism as saw palmetto — weak 5α-reductase inhibition; phytosterols are concentrated in pumpkin seed oil.

  • β-SitosterolPhytosterol

    Same primary phytosterol as saw palmetto; weak 5α-reductase inhibition with anti-inflammatory effects on prostate tissue.

  • β-sitosterol + lectin (UDA)phytosterol + glycoprotein

    Inhibit conversion of testosterone to dihydrotestosterone (DHT) and bind to sex-hormone-binding globulin in vitro; the principal mechanism for the modest BPH symptom relief observed with nettle root extract.

A shared molecular target shows how a botanical and a drug relate mechanistically. It is not evidence that one can replace the other. Educational summary only — discuss any medication decision with your clinician.