Molecular target
β1-Adrenergic Receptor
Predominantly cardiac adrenergic receptor; antagonism reduces heart rate, contractility, and renin release. Targeted by cardioselective beta-blockers (metoprolol, atenolol, bisoprolol).
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Pharmaceutical agents
Drugs that act on β1-Adrenergic Receptor
These medications have β1-Adrenergic Receptor among their molecular targets. Sharing a target is a mechanistic relationship — it does not make any plant below an alternative to, or substitute for, these drugs.
Botanical connections
Plants whose compounds act on β1-Adrenergic Receptor
Each plant below contains a named compound documented to act on β1-Adrenergic Receptor. The compound and the reason for the connection are shown on every edge — a shared mechanism, not a therapeutic equivalence.
- Ephedrinephenethylamine alkaloid
Indirect-acting sympathomimetic (releases norepinephrine from sympathetic nerve terminals) plus direct α1, β1, β2 adrenergic agonism. The combination produces vasoconstriction, increased cardiac output, and bronchodilation — therapeutic for bronchospasm and decongestion but at significant cardiovascular cost.
- Pseudoephedrinephenethylamine stereoisomer
Stereoisomer of ephedrine with greater nasal-decongestant selectivity — the basis for FDA-permitted single-ingredient pseudoephedrine OTC products (Sudafed). Lower cardiovascular impact than ephedrine but still meaningful at high dose.