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MytoIntelligence
All targets

Molecular target

Serotonin Transporter

Also: 5-HTT, SLC6A4 · id SERT

Reuptake transporter that clears serotonin from the synaptic cleft. The primary pharmacological target of SSRIs.

28 drugs act here5 plants reach it via their compounds

Educational use only. This page summarizes published research and traditional-use records for educational purposes. It does not diagnose, treat, cure, or prevent any disease. Do not start, stop, or change medications based on this information. Discuss any decisions about therapies — pharmaceutical or botanical — with a qualified clinician who knows your medical history.

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Pharmaceutical agents

Drugs that act on Serotonin Transporter

These medications have Serotonin Transporter among their molecular targets. Sharing a target is a mechanistic relationship — it does not make any plant below an alternative to, or substitute for, these drugs.

Botanical connections

Plants whose compounds act on Serotonin Transporter

Each plant below contains a named compound documented to act on Serotonin Transporter. The compound and the reason for the connection are shown on every edge — a shared mechanism, not a therapeutic equivalence.

  • MesembrineMesembrine-type alkaloid

    High-affinity selective serotonin reuptake inhibitor with nanomolar SERT binding — pharmacologically comparable to known SSRIs. Basis for the HIGH-severity interaction warning with prescription SSRIs/SNRIs/MAOIs. Optimized in the Trimesemine™ standardization for serotonergic support without the cytotoxicity concerns of Δ7-rich extracts.

  • MesembrenoneMesembrine-type alkaloid

    Dual mechanism — selective serotonin reuptake inhibitor PLUS phosphodiesterase-4 (PDE4) inhibitor. PDE4 inhibition raises intracellular cAMP, supporting neuroplasticity and memory. Dominant alkaloid in the Zembrin® standardized extract (mesembrenone + mesembrenol = >60% of profile). Terburg 2013 demonstrated reduced amygdala-hypothalamus coupling on fMRI with Zembrin.

  • Δ7-Mesembrenone (Delta-7-Mesembrenone)Mesembrine-type alkaloid (delta-isomer)

    Potent antioxidant and reactive-oxygen-species scavenger with neuroprotective and antiproliferative activity in vitro — but with dose-dependent cytotoxicity in cultured astrocytes (narrow safety margin). Bennett 2018 compared Δ7-rich extracts to Trimesemine™. Cytotoxicity profile is the basis for favoring standardized Zembrin® or Trimesemine™ over Δ7-enriched preparations of unknown chemotype.

  • IbogaineIndole alkaloid (iboga-type)

    Multi-target alkaloid — proposed μ-opioid antagonist or partial agonist, κ-opioid agonist, NMDA antagonist, SERT inhibitor, σ receptor binding, neurotrophic effects via GDNF upregulation. The 'visioning' phase of an ibogaine session lasts 12-36 hours. **QT prolongation** is the dominant safety concern.

  • Noribogaine (active metabolite)Indole alkaloid

    Hepatically demethylated metabolite with longer half-life than parent ibogaine; thought to mediate the post-acute mood-stabilizing and craving-reducing phase of ibogaine therapy.

  • SafranalMonoterpene aldehyde

    Volatile aroma compound; in vitro SERT inhibition and 5-HT1A modulation contribute to antidepressant signal.

  • HyperforinPhloroglucinol

    Primary antidepressant constituent; inhibits SERT, NET, and DAT through a non-classical mechanism (TRPC6-mediated). Major source of CYP3A4 induction → drug-interaction profile.

  • Triterpene glycosides (actein, cimicifugoside)Triterpene glycoside

    Modern mechanism understanding: serotonergic activity, NOT estrogen receptor binding. The confusion arose because the indication overlap with phytoestrogens (menopausal symptoms) was historically assumed to imply mechanism overlap — modern binding studies show no ER affinity.

A shared molecular target shows how a botanical and a drug relate mechanistically. It is not evidence that one can replace the other. Educational summary only — discuss any medication decision with your clinician.